CALICIVIRUS EN HUMANOS PDF

Thirteen outbreak strains sequenced were noroviruses, including 8 GII, 2 GI, and 3 belonging to a novel genogroup. Human caliciviruses HuCVs , especially noroviruses, are a major cause of food- and waterborne outbreaks in industrialized countries. Their role as a cause of gastroenteritis outbreaks in economically developing areas is unclear because little information is available 1 — 3. Strains differing significantly from GI and GII prototypes are being increasingly reported since detection methods have improved 5 , 6. Chile is a rapidly developing country.

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Investigador Cinvestav 3C. Fax: 33 77 E-mail: alonso cinvestav. Our group is interested in the study of calicivirus host-cell interactions, particularly in the role of cellular proteins in viral replication and during apoprotis induction during infection.

Since there is not a convenient cell culture to propagate human caliciviruses HuCVs , we are using two surrogate models: feline calicivirus FCV and murine norovirus MNV to study calicivirus biology. On the other hand, we are also studding the molecular epidemiology of HuCVs, through its detection in feces, with the propose of knowing the incidence of this viruses in our population.

Sauceda, R. Novel combination of c-myc, n-myc and n-ras oncogene alteration in brain tumors. Molecular Brain Reserach. Nuclear proteins bind to poliovirus 5' untranslated region.

Archives of Medical Research. Attenuating mutations in the poliovirus 5' untranslated region alter its interaction with polypirimidine tract-binding protein. Journal of Virology. Differences in the uv-crosslinking patterns of the poliovirus 5' untranslated region with cell proteins from poliovirus susceptible and resistant tissues.

Interaction of cellular proteins with the 5' end of Norwalk virus genomic RNA. Biochemical and Biophysical Research Communications. Journal of Virological Methods. The trichomonad cystein proteinase TVCP4 transcript contains iron-responsive element. FEBS Letters. Human Caliciviruses detected in Mexican children admitted to hospital during , with severe acute gastroenteritis not due to other enteropathogens. Journal of Medical Virology. Cancio-Lonches C. Tolentino-Ruiz, R. Development of a Multiplex PCR assay to detect gastroenteric pathogens in the feces of mexican children.

Current Microbiology. Vol Dusetty, P. Evaluation of the second generation of a comercial latex agglutination test for the detection of rotavirus antigens in fecal samples. Host-cell factors involved in the calicivirus replicative cycle. Future Virology. Vol 9 2 Escalera, Cueto, M. Overexpression of let-7c miRNA inhibits dengue virus replication in human hepatoma Huh-7 cells. Virus Research. Nucleolin promotes in vitro translation of feline calicivirus genomic RNA.

Taboada, B. The geographic structure of. Environ Microbiol. Annexin A2 associates to feline calicivirus RNA in the replication complexes from infected cells and participates in an efficient viral replication. The feline calicivirus leader of the capsid protein causes survivin and XIAP downregulation and apoptosis. Vol 25, pp Estrategias replicativas del virus Norwalk. Avance y Perspectiva. Vol 1 2 Noviembre,

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Investigador Cinvestav 3C. Fax: 33 77 E-mail: alonso cinvestav. Our group is interested in the study of calicivirus host-cell interactions, particularly in the role of cellular proteins in viral replication and during apoprotis induction during infection. Since there is not a convenient cell culture to propagate human caliciviruses HuCVs , we are using two surrogate models: feline calicivirus FCV and murine norovirus MNV to study calicivirus biology. On the other hand, we are also studding the molecular epidemiology of HuCVs, through its detection in feces, with the propose of knowing the incidence of this viruses in our population. Sauceda, R. Novel combination of c-myc, n-myc and n-ras oncogene alteration in brain tumors.

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Caliciviruses and Foodborne Gastroenteritis, Chile

Scaravelli, Mendoza, Argentina. Miguel L. Human caliciviruses HuCVs are increasingly recognized as common pathogens that cause acute sporadic diarrhea in children; however, regional antigenic and genetic diversity complicate detection techniques. Stool samples from children seeking medical attention in 2 outpatient clinics, a large emergency department, and 2 hospital wards were evaluated for HuCVs by reverse transcription—polymerase chain reaction, using primers based on a conserved sequence of the polymerase region of a previously sequenced Chilean strain. The decline may have been due to a change in virus genotype.

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Em , Rossignol 40 analisou um novo medicamento indicado para o tratamento de diarreia causada por gastroenterite viral, chamado Nitazoxanide. De a , ocorreram surtos de NoV na Espanha. Na Inglaterra, Lopman et al. Chapin et al.

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